[1]Zenker J, Ziegler D, Chrast R. Novel pathogenic pathways in diabetic neuropathy[J]. Trends Neurosci, 2013, 36(8): 439-449.
[2]Feldman EL, Callaghan BC, Pop-Busui R, et al. Diabetic neuropathy[J]. Nat Rev Dis Primers, 2019, 5(1): 41-59.
[3]Chandrasekaran K, Anjaneyulu M, Choi J, et al. Role of mitochondria in diabetic peripheral neuropathy: Influencing the NAD-dependent SIRT1-PGC-1α-TFAM pathway[J]. Int Rev Neurobiol, 2019, 145: 177-209.
[4]Yagihashi S, Yamagishi SI, Wada Ri R, et al. Neuropathy in diabetic mice overexpressing human aldose reductase and effects of aldose reductase inhibitor[J]. Brain, 2001, 124(Pt 12): 2448-2458.
[5]Dewanjee S, Das S, Das A, et al. Molecular mechanism of diabetic neuropathy and its pharmacotherapeutic targets[J]. Eur J Pharmacol, 2018, 833: 472-523.
[6]Chang YS, Kan HW, Hsieh YL. Activating transcription factor 3 modulates protein kinase C epsilon activation in diabetic peripheral neuropathy[J]. J Pain Res, 2019, 12: 317-326.
[7]Verge VMK, Andreassen CS, Arnason TG, et al. Mechanisms of disease: role of neurotrophins in diabetes and diabetic neuropathy[J]. Handb Clin Neurol, 2014, 126: 443-460.
[8]李海斌, 杨生健, 蔡春茂, 等. 甲钴胺和银杏叶联合治疗老年糖尿病周围神经病变22例[J]. 东南国防医药, 2011, 13(4): 362.
[9]Pang L, Lian X, Liu HQ, et al. Understanding Diabetic Neuropathy: Focus on Oxidative Stress[J]. Oxid Med Cell Longev, 2020, 2020:9524635.
[10]Liu YP, Shao SJ, Guo HD. Schwann cells apoptosis is induced by high glucose in diabetic peripheral neuropathy[J]. Life Sci, 2020, 248: 117459.
[11]Schwarz DS, Blower MD. The endoplasmic reticulum: structure, function and response to cellular signaling[J]. Cell Mol Life Sci, 2016, 73(1): 79-94.
[12]Márton M, Kurucz A, Lizák B, et al. A Systems Biological View of Life-and-Death Decision with Respect to Endoplasmic Reticulum Stress-The Role of PERK Pathway[J]. Int J Mol Sci, 2017, 18(1): 58.
[13]Deegan S, Koryga I, Glynn S, et al. A close connection between the PERK and IRE arms of the UPR and the transcriptional regulation of autophagy[J]. Biochem Biophys Res Commun, 2015, 456(1): 305-311.
[14]O′Brien P, Hinder L, Sakowski S, et al. ER stress in diabetic peripheral neuropathy: A new therapeutic target[J]. Antioxid Redox Signal, 2014, 21(4): 621-633.
[15]Lupachyk S, Watcho P, Stavniichuk R, et al. Endoplasmic reticulum stress plays a key role in the pathogenesis of diabetic peripheral neuropathy[J]. Diabetes, 2013, 62(3): 944-952.
[16]Ariyasu D, Yoshida H, Hasegawa Y. Endoplasmic Reticulum (ER) Stress and Endocrine Disorders[J]. Int J Mol Sci, 2017, 18(2): 382.
[17]Gundu C, Arruri V, Sherkhane B, et al. Indole-3-propionic acid attenuates high glucose induced ER stress response and augments mitochondrial function by modulating PERK-IRE1-ATF4-CHOP signalling in experimental diabetic neuropathy[J]. Arch Physiol Biochem, 2022, 1-14.
[18]El-Horany H, Watany M, Hagag R, et al. Expression of LRP1 and CHOP genes associated with peripheral neuropathy in type 2 diabetes mellitus: Correlations with nerve conduction studies[J]. Gene, 2019, 702: 114-122.
[19]Pan H, Huang H, Zhang L, et al. "Adjusting internal organs and dredging channel" electroacupuncture treatment prevents the development of diabetic peripheral neuropathy by downregulating glucose-related protein 78 (GRP78) and caspase-12 in streptozotocin-diabetic rats[J]. J Diabetes, 2019, 11(12): 928-937.
[20]Song B, Scheuner D, Ron D, et al. Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes[J]. J Clin Invest, 2008, 118(10): 3378-3389.
[21]Yang X, Yao W, Liu H, et al. Tangluoning, a traditional Chinese medicine, attenuates in vivo and in vitro diabetic peripheral neuropathy through modulation of PERK/Nrf2 pathway[J]. Sci Rep, 2017, 7(1): 1014-1025.
[22]Clark A, Urano F. Endoplasmic reticulum stress in beta cells and autoimmune diabetes[J]. Curr Opin Immunol, 2016, 43: 60-66.
[23]Yao W, Yang X, Zhu J, et al. IRE1α siRNA relieves endoplasmic reticulum stress-induced apoptosis and alleviates diabetic peripheral neuropathy in vivo and in vitro[J]. Sci Rep, 2018, 8(1): 2579-2591.
[24]Madhusudhan T, Wang H, Dong W, et al. Defective podocyte insulin signalling through p85-XBP1 promotes ATF6-dependent maladaptive ER-stress response in diabetic nephropathy[J]. Nat Commun, 2015, 6: 6496-6511.
[25]Tabassum R, Jeong NY, Jung J. Protective effect of hydrogen sulfide on oxidative stress-induced neurodegenerative diseases[J]. Neural Regen Res, 2020, 15(2): 232-241.
[26]Sadeghiyan Galeshkalami N, Abdollahi M, Najafi R, et al. Alpha-lipoic acid and coenzyme Q10 combination ameliorates experimental diabetic neuropathy by modulating oxidative stress and apoptosis[J]. Life Sci, 2019, 216: 101-110.
[27]熊燕. α-硫辛酸联合依帕司他治疗糖尿病周围神经病变临床疗效的Meta分析[J]. 东南国防医药, 2017, 19(1): 58-61.
[28]Figueroa-Romero C, Sadidi M, Feldman E. Mechanisms of disease: the oxidative stress theory of diabetic neuropathy[J]. Rev Endocr Metab Disord, 2008, 9(4): 301-314.
[29]Li Q, Wang Z, Zhou W, et al. Epalrestat protects against diabetic peripheral neuropathy by alleviating oxidative stress and inhibiting polyol pathway[J]. Neural Regen Res, 2016, 11(2): 345-351.
[30]Soltesova Prnova M, Ballekova J, Gajdosikova A, et al. A novel carboxymethylated mercaptotriazinoindole inhibitor of aldose reductase interferes with the polyol pathway in streptozotocin-induced diabetic rats[J]. Physiol Res, 2015, 64(4): 587-591.
[31]Oates PJ. Aldose Reductase, Still a Compelling Target for Diabetic Neuropathy[J]. Curr Drug Targets, 2008, 9(1): 14-36.
[32]Geicu OI, Stanca L, Voicu SN, et al. Dietary AGEs involvement in colonic inflammation and cancer: insights from an in vitro enterocyte model[J]. Sci Rep, 2020, 10(1): 1-14.
[33]Wu TT, Chen YY, Chang HY, et al. AKR1B1-Induced Epithelial-Mesenchymal Transition Mediated by RAGE-Oxidative Stress in Diabetic Cataract Lens[J]. Antioxidants, 2020, 9(4): 273.
[34]Shakeel M. Recent advances in understanding the role of oxidative stress in diabetic neuropathy[J]. Diabetes & metabolic syndrome, 2015, 9(4): 373-378.
[35]Babizhayev MA, Strokov IA, Nosikov VV, et al. The Role of Oxidative Stress in Diabetic Neuropathy: Generation of Free Radical Species in the Glycation Reaction and Gene Polymorphisms Encoding Antioxidant Enzymes to Genetic Susceptibility to Diabetic Neuropathy in Population of Type I Diabetic Patients[J]. Cell Biochem Biophys, 2015, 71(3): 1425-1443.
[36]Li X, Zhao Z, Kuang P, et al. Regulation of lipid metabolism in diabetic rats by Arctium lappa L. polysaccharide through the PKC/NF-κB pathway[J]. Int J Biol Macromol, 2019, 136: 115-122.
[37]Kishore L, Kaur N, Singh R. Effect of Kaempferol isolated from seeds of Eruca sativa on changes of pain sensitivity in Streptozotocin-induced diabetic neuropathy[J]. Inflammopharmacology, 2017, 26(4): 1-11.
[38]Koroglu P, Bulan OK, Ozakpinar OB, et al. The effect of oxytocin treatment on the adult liver of streptozotocin (STZ) - Induced diabetic rats[J]. Fresenius Environ Bull, 2017, 26(8): 5013-5020.
[39]Simmen T, Lynes EM, Gesson K, et al. Oxidative protein folding in the endoplasmic reticulum: tight links to the mitochondria-associated membrane (MAM)[J]. Biochim Biophys Acta, 2010, 1798(8): 1465-1473.
[40]Maamoun H, Benameur T, Pintus G, et al. Crosstalk Between Oxidative Stress and Endoplasmic Reticulum (ER) Stress in Endothelial Dysfunction and Aberrant Angiogenesis Associated With Diabetes: A Focus on the Protective Roles of Heme Oxygenase (HO)-1[J]. Front Physiol, 2019, 10: 70.
[41]Lewis SC, Uchiyama LF, Nunnari J. ER-mitochondria contacts couple mtDNA synthesis with mitochondrial division in human cells[J]. Science, 2016, 353(6296): aaf5549.
[42]Yang X, Yao W, Liu H, et al. Tangluoning, a traditional Chinese medicine, attenuates in vivo and in vitro diabetic peripheral neuropathy through modulation of PERK/Nrf2 pathway[J]. Sci Rep, 2017, 7(1): 1014.
[43]Jin JK, Blackwood EA, Azizi KM, et al. ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart[J]. Circ Res, 2016, 120(5): 862-875.
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